Passive smoking - 136 entries found

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1990

CIS 91-1594 Cummings K.M., Markello S.J., Mahoney M., Bhargava A.K., McElroy P.D., Marshall J.R.
Measurement of current exposure to environmental tobacco smoke
An epidemiological study was performed in a cancer screening clinic in Buffalo (NY, USA) in 1986 to determine the effects of exposure to environmental tobacco smoke among 663 never- and ex-smokers by using measurement of urinary cotinine levels and determining the prevalence of exposure to environmental tobacco smoke. The most frequently mentioned sources of exposure to environmental tobacco smoke were at work (28%) and at home (27%). Cotinine was found in the urine of 91% subjects. Cotinine values increased significantly with the number of exposures reported.
Archives of Environmental Health, Mar.-Apr. 1990, Vol.45, No.2, p.74-79. Illus. 15 ref.

CIS 91-1160 Woodward S.D., Winstanley M.H.
Lung cancer and passive smoking at work: the Carroll case
Compensation cases indicating environmental tobacco smoke as a cause of illness have been tested in Australian courts. Mr. Sean Carroll, a Melbourne bus driver, was awarded AUD 65,000 in an out-of-court settlement as compensation for developing small cell carcinoma of the lung, probably as a result of exposure to his passengers' and co-workers' tobacco smoke over the 35 years of his employment. It was estimated that there was a 75% probability that Carroll's lung cancer was caused by passive smoking, and that any contribution by air pollution or diesel fumes was marginal.
Medical Journal of Australia, 3/17 Dec. 1990, Vol.153, No.11/12, p.682-684. 18 ref.

CIS 91-837 Brugnone F., Perbellini L., Maranelli G., Romeo L., Alexopoulos C., Gobbi M.
Effects of cigarette smoking on blood and alveolar levels of benzene
Effetto del fumo di sigaretta sui livelli ematici ed alveolari di benzene [in Italian]
Benzene was measured in the blood and alveolar air of 34 chemical industry workers exposed to benzene and 134 controls. The results of the biological measurements were compared with the environmental levels of benzene in the room where samples were taken and at the worksite during the previous day's shift. All environmental air samples showed measurable levels of benzene, which agrees with previous observations showing that benzene is a common pollutant also of the living and external environments. In the controls, both blood and alveolar concentrations of benzene were significantly higher in the 68 smokers than in the 66 non-smokers and the biological levels of benzene were inversely correlated with the time that had elapsed since the last cigarette smoked. In the chemical workers, the high biological levels of benzene due to occupational exposure largely exceeded the variations in concentration due to cigarette smoking and cancelled out the differences between smokers and non-smokers. Thus, smoking did not interfere with the interpretation of the results in the occupationally exposed workers, but as a cyclical and additional factor of exposure, it was responsible for the disturbance in the relationships between biological benzene levels and ubiquitous environmental pollution.
Medicina del lavoro, Mar.-Apr. 1990, Vol.81, No.2, p.101-106. Illus. 28 ref.

CIS 91-955 Limit values for concentrations of dangerous substances in workplace air: USA (ACGIH), FRG and USSR
Valeurs limites d'exposition professionnelle aux substances dangereuses de l'ACGIH, de la RFA et de l'URSS [in French]
This data sheet presents the limit values adopted in the US (American Conference of Governmental Industrial Hygienists - ACGIH) (values for 1989-1990); in the Federal Republic of Germany (Deutsche Forschungsgemeinschaft - DFG) and in the USSR (new standard GOST 12.1.005-88 of 1988). More than 1600 chemical substances are listed. The appendix includes further information drawn either from American (carcinogenic substances, mixtures, special working hours, dangerous substances with varying composition, dusts, proposals for modifying limit values or adding to the list) or from West German sources (carcinogenic substances, technical guiding concentrations (TRK), mixtures, limit values and pregnancy, mutagens, metals and metal compounds, occupational passive smoking, dusts).
Cahiers de notes documentaires - Sécurité et hygiène du travail, 1st Quarter 1990, No.138, Note No.1770-138-90, p.135-189. Annexes.

CIS 90-1956 Kasuga H.
Indoor air quality
Papers presented at the International Conference on Indoor Air Quality, 4-6 Nov. 1987, in Tokyo, Japan. Keynote lectures on passive smoking are followed by presentations grouped under the headings "environmental tobacco smoke measurement", "biological effects associated with exposure to environmental tobacco smoke", "epidemiology of passive smoking" and "general indoor air pollution".
Springer-Verlag, 17 Tiergartenstrasse, W-6900 Heidelberg 1, Germany, 1990. 529p. Illus. Bibl. Price: DEM 198.00.

CIS 90-709 Tolerable threshold limits at the workplace 1990: Substances hazardous to health (MAV values); Physical agents [Switzerland]
Valeurs limites d'exposition aux postes de travail 1990: substances dangereuses pour la santé (VME/VLE); agents physiques [Suisse] [in French]
Definitions of TLVs for the concentration of toxic substances at the workplace; list of MAC values for 1990 (200 showing substance names, chemical formulae, values expressed both in mL/m³ (ppm) and in mg/m³, with remarks on the source when appropriate). In the appendix: carcinogens, organic peroxides, inert substances, dust and gases, passive smoking, infrared radiation and carcinogenic nitroamines. This list includes information on potential biological monitoring of substances for which biological threshold values might be established.
Caisse nationale suisse d'assurance en cas d'accidents, Case postale, 6002 Lucerne, Suisse, 1990. 125p.

1989

CIS 90-1655 Price P.
Passive smoking. Health effects and workplace resolutions
Le tabagisme passif - Effets sur la santé et solutions en milieu de travail [in French]
Passive smoking (also known as involuntary smoking) is the exposure of non-smokers to tobacco smoke. The tobacco smoke inhaled by passive smokers is usually called environmental tobacco smoke. Concern about its health effects is relatively recent and parallels evidence of health effects in non-smokers. The available information is summarised here as: tobacco smoke composition and exposure; signs, symptoms and possible consequences of exposure; ventilation standards and requirements for tobacco smoke; policies and programmes in the workplace.
Canadian Centre for Occupational Health and Safety, 250 Main Street East, Hamilton, Ontario L8N 1H6, Canada, Feb. 1989. 9p. 23 ref.

CIS 90-928 Kentner M., Weltle D., Valentin H.
Passive smoking and pulmonary function in adults
Passivrauchen und Lungenfunktion bei Erwachsenen [in German]
Study of the effects of passive smoking exposure on lung function. A group of 1,364 office employees free of lung diseases were divided into smokers, non-smokers, passive smokers and ex-smokers. Lung function parameters such as forced vital capacity, peak expiratory flow and maximal expiratory flow rates were determined. Results were standardised for age, height and body weight. No essential lung function impairments were found for passive smokers.
Arbeitsmedizin - Sozialmedizin - Präventivmedizin, 1989, Vol.24, No.1, p.8-13. Illus. 62 ref.

CIS 90-213 Kristensen T.S.
Cardiovascular disorders and the work environment - A critical review of the epidemiologic literature on chemical factors
This is the second of two articles reviewing the epidemiologic research on cardiovascular diseases (CVD) and the work environment. It deals with chemical factors, i.e. lead, cadmium, cobalt, arsenic, carbon monoxide, passive smoking, organic solvents, carbon disulfide, nitroglycerin, nitroglycol, and others. The epidemiologic literature relating to each is assessed on the basis of a number of methodological criteria, and the need for future research, the methodology of literature reviews, and preventive implications and perspectives are discussed. It is concluded that the causal relationship between two of the chemicals, carbon disulfide and nitroglycerin/nitroglycol, and CVD is very well documented. For lead and passive smoking a causal relation to CVD is likely. More research is needed concerning cobalt, arsenic, antimony, and other chemical compounds. Exposure to carbon monoxide increases the acute risk of CVD but has probably no lasting atherosclerotic effect. Cadmium and organic solvents are probably not causally related to CVD.
Scandinavian Journal of Work, Environment and Health, Aug. 1989, Vol.15, No.4, p.245-264. 296 ref.

CIS 90-182 Robins J.M., Blevins D., Schneiderman M.
The effective number of cigarettes inhaled daily by passive smokers: Are epidemiologic and dosimetric estimates consistent?
Since the early 1980's, a number of epidemiologic studies have implicated environmental tobacco smoke (ETS) as a cause of lung cancer among non-smokers passively exposed to other people's tobacco smoke. A recent National Academy of Science Report (USA) on environmental tobacco smoke (NAS, 1986) summarised 13 such studies. Each study provided an estimate of the ratio of the lung cancer mortality rate among non-smokers with smoking spouses to the mortality rate among those with non-smoking spouses. The weighted average of the 13 study-specific rate ratios was roughly 1.3. It is shown that if this summary rate ratio is causally related to ETS and not to bias then the estimated number of lung cancer deaths attributable to ETS exposure occurring in US non-smokers in 1985 lies in the range of 2500-5000. Further it is examined whether the summary rate ratio of 1.3 is consistent with the existing epidemiologic data on active smokers and the dosimetric measurements that have been made on mainstream and environmental tobacco smoke. If consistent with this other data, the hypothesis is that the rate ratio of 1.3 is causally related to ETS exposure will be strengthened.
Journal of Hazardous Materials, May 1989, Vol.21, No.3, p.215-238. 18 ref.

CIS 89-1959 Meili B.
Protection of non-smokers in the workplace
Nichtraucherschutz am Arbeitsplatz [in German]
Measures are proposed to convince employees within enterprises to stop smoking or at least to reduce their cigarette consumption.
Illustrierte Zeitschrift für Arbeitsschutz, Apr. 1989, Vol.39, No.2, p.13-16.

CIS 89-1829 Blyth-Bristow B.
Introducing a smoking policy at work - A case study
The article describes the step-by-step introduction of a new smoking policy in the Performing Right Society (London), which resulted in setting up a smoke-free working environment. Two main objectives of anti-smoking policy - education of workers about the harmful effects of smoking and change of the working environment in order to minimise the harmful effects of passive smoking on the non-smoker are pointed out.
Occupational Health, June 1989, Vol.41, No.6, p.163-165.

1988

CIS 91-588
Health and Safety Executive
Passive smoking at work
This booklet dealing with passive smoking suggests ways of reducing the exposure of non-smokers to tobacco smoke at work, and answers some common questions about risks to the health of non-smokers from tobacco smoke.
Health and Safety Executive, Library and Information Services, Broad Lane, Sheffield S3 7HQ, United Kingdom, 1989. 9p.

CIS 91-423 Passive smoking at work
This booklet provides information about passive smoking at work. It suggests ways of reducing the exposure of non-smokers to tobacco smoke at work, and answers some common questions about risks to the health of non-smokers from tobacco smoke. Contact addresses for further information are provided.
Health and Safety Executive, Library and Information Services, Broad Lane, Sheffield S3 7HQ, United Kingdom, 1988. 10p.

CIS 90-244 Sterling T.D., Mueller B.
Concentrations of nicotine, RSP, CO and CO₂ in nonsmoking areas of offices ventilated by air recirculated from smoking designated areas
Concentration of nicotine, respirable suspended particles (RSP), carbon monoxide (CO), and carbon dioxide (CO₂) were measured in offices under different conditions of smoking regulation: smoking prohibited; smoking prohibited areas receiving recirculated air from designated smoking areas; smoking and nonsmoking sections of these designated smoking areas. Nicotine levels in non-smoking receiving recirculated air from smoking offices were approximately 1.0µg/m³. RSP, CO and CO₂ concentrations were approximately the same in these offices as compared to nonsmoking offices not exposed to recirculated air from smoking areas. Providing a designated but not separately ventilated smoking area appears to be effective in eliminating most components of ETS from nonsmoking office work areas.
American Industrial Hygiene Association Journal, Sep. 1988, Vol.49, No.9, p.423-426. 7 ref.

CIS 89-1273 Vaskövi B., Szívós K., Kertész M., Horváth A., Bácskai G.
Role of tobacco smoking in interior cadmium exposure
A dohányzás szerepe a belsőtéri kadmium-expozícióban [in Hungarian]
The cadmium (Cd) content of 4 different types of Hungarian-made cigarettes was measured by atomic absorption spectrometry. The measured values were 0.46-1.64µg/g. The aerosol concentration of smoke resulting from the simultaneous burning of 6 cigarettes was also evaluated in a room with cubic capacity of 14m³. This aerosol concentration decreased exponentially with time, reaching approx. background levels after 2h. Cd contamination of the air in the room was in the range of 0.033-0.100µg/m³, a level that would contribute significantly to the total Cd exposure of people working in rooms of similar size, including that of non-smokers (passive smoking).
Egészségtudomány, 1988, Vol.32, p.14-19. 17 ref.

CIS 89-965 Limit values for concentrations of dangerous substances in workplace air: USA (ACGIH), FRG and USSR
Valeurs limites d'exposition professionnelle aux substances dangereuses de l'ACGIH, de la RFA et de l'URSS [in French]
This data sheet presents the limit values adopted in the US (American Conference of Governmental Industrial Hygienists - ACGIH), in the Federal Republic of Germany (Deutsche Forschungsgemeinschaft - DFG) and in the USSR (GOST Standard). More than 1600 chemical substances are listed. In the appendix, further information is included, drawn either from American sources (carcinogenic substances, mixtures, dangerous substances with varying composition, dusts, proposals for modifying limit values or adding to the list) or West German sources (carcinogenic substances, technical guiding concentrations (TRK), limit values and pregnancy, metals and metal compounds, occupational passive smoking, dusts).
Cahiers de notes documentaires - Sécurité et hygiène du travail, 4th. Quarter 1988, No.133, Note No. 1709-133-88, p.707-753.

CIS 88-1750 The effects of passive smoking
This data sheet covers: evidence for the effects of passive smoking; smoking policies in the workplace; success of a policy.
United Trade Press Limited, 33-35 Bowling Green Lane, London EC1R ODA, United Kingdom, 1988. 5p. 7 ref.

1987

CIS 89-1270 Armstrong B.K.
Commentary: Passive smoking and lung cancer
This article reviews several studies carried out in various countries in order to examine the relationship between passive smoking and respiratory disorders. The results show that passive smoking leads to reduction of lung function and gives rise to an increased risk of cancer. An estimate is provided on the number of deaths per year in Australia due to passive smoking (136).
Community Health Studies, Supplement to Vol.XI, No.1, 1987, p.6-8. 10 ref.

CIS 89-1158 Information on health and safety hazards in hotels, restaurants, and catering establishments
Training guide covering the various risks affecting workers in the hotel and restaurant industry. Main topics covered: VDUs; stress; work in bars; cellar machinery and equipment; asbestos; food hygiene; kitchen design, equipment and machinery; microwave ovens; pest control; chemicals; temperature; ventilation; electrical equipment; noise; lighting; manual lifting; shiftwork; women workers; fires; passive smoking.
International Union of Food and Allied Workers (IUF), Rampe du Pont-Rouge 8, 1213 Petit-Lancy, Genève, Switzerland, May 1987. 26p. Illus.

CIS 89-700 Jenkins M., McEwen J., Moreton J., East R., Seymour L., Goodin M.
Smoking policies at work
This booklet describes the problems which may arise from smoking at work and recommends possible policies in response to an increasing number of demands from both smokers and non-smokers concerning the matter.
Health Education Authority, 78 New Oxford Street, London WC1A 1AH, United Kingdom, 1987. 48p. Illus. Bibl. ref.

CIS 88-1477
Al-ma ⁽had ul-⁽arabiyy li ṣ-ṣiḥḥa wa-s-salâmat il-mihniyya
Safety and health posters
Set of 8 posters to encourage maintenance of electrical equipment, use of hearing protectors, reading of instructions before using or storing chemical products, use of warning signs and flagmen during work on roads and proper lifting and carrying techniques, and to discourage smoking (control passive smoking), careless work and carelessness with fire.
Arab Institute for Occupational Health and Safety, P.O. Box 5770, Damascus, Syria, no date. 8 posters.

CIS 88-1250 Wallace L., Pellizzari E., Hartwell T.D., Ziegenfus R.
Exposures to benzene and other volatile compounds from active and passive smoking
Personal exposures and breath concentrations of approximately 20 volatile organic substances were measured for 200 smokers and 322 nonsmokers in New Jersey and California. Smokers displayed significantly elevated breath levels of benzene, styrene, ethylbenzene, m+p-xylene, o-xylene, and octane. Significant increases in breath concentration with number of cigarettes smoked were noted for the first four aromatic compounds. Based on direct measurements of benzene in mainstream cigarette smoke, it is calculated that a typical smoker inhales 2mg benzene daily, compared to 0.2mg/day for the nonsmoker. Passive smokers exposed at work had significantly elevated levels of aromatics in their breath.
Archives of Environmental Health, Sep.-Oct. 1987, Vol.42, No.5, p.272-279. Illus. 20 ref.

CIS 87-1288 Vainio H.
Is passive smoking increasing cancer risk ?
The many published epidemiologic studies are all consistent with a 30% increase in the risk of lung cancer. This increase is plausible in relation to the exposure levels derived from various biological dose indicators. The risks of passive smokers are, of course, smaller than those of active smokers, but it is generally accepted that involuntary risks should be much smaller than those that are self-inflicted. Even a relative risk for lung cancer of 1.3 due to passive smoking would constitute an increase of 390-990 per 100,000 deaths in the life-time risk of dying of lung cancer in people who have never smoked. Normally, this level would be considered "unacceptable", and preventive measures should be taken.
Scandinavian Journal of Work, Environment and Health, June 1987, Vol.13, No.3, p.193-196. 36 ref.

1986

CIS 89-350 IARC monographs on the evaluation of the carcinogenic risk of chemicals to humans - Tobacco smoking
Conclusions of a meeting of experts (Lyon, France, Feb. 1986) on public health. Contents: worldwide tobacco-smoking patterns; chemistry and analysis of smoke; biological data relevant to the evaluation; epidemiological studies of cancer in humans; conclusions and evaluations; international tobacco sales, 1966-1983.
World Health Organization, Distribution and Sales Service, 1211 Genève 27, Switzerland, 1986. 421p. Illus. Bibl.

CIS 88-1602 Olander L., Johansson J.
Measurement of cigarette smoke during passive smoking - Report on particle concentration measurements under controlled conditions
Mätning av cigarettrök vid passiv rökning - Redovisning av partikelhaltsmätningar under kontrollerade betingelser [in Swedish]
Exposure of non-smokers to cigarette smoke, carbon monoxide and particle concentrations was measured. Aerosol size distributions and concentrations for the interval 0.01-1.0µm were measured in an exposure room of 100m³ during exposures to smoke from 5, 10 and 20 cigarettes during 1, 2 and 4h. The significance of the measured concentrations for the exposure is discussed. A comparison with measured concentrations in body fluids will be made in a separate report.
Arbetarskyddsstyrelsen, Publikationsservice, 171 84 Solna, Sweden, 1986. 43p. Illus. 13 ref.

CIS 88-276 Henschler D.
Harmful industrial substances - Toxicological and medical basis for the establishment of maximum permissible concentrations
Gesundheitsschädliche Arbeitsstoffe - Toxikologisch-arbeitsmedizinische Begründung von MAK-Werten [in German]
This loose-leaf collection was published under the auspices of the Toxic Substance Control Committee of the German Research Association. It contains basic information on approx. 300 substances (chemicals, gases, fumes, silica, asbestos, man-made mineral fibres, cutting fluids). Supplement 11 had one chapter devoted to passive smoking. Supplement 12 provides additional studies on: aluminium fumes and dust; acetaldehyde (update); ammonia (update); 1,4 and 2,4-butanesultone; 2-methyl-4-chloroaniline; dichloromethane (update); 3-3'-dimethoxybenzidine; 3-3'-dimethylbenzidine; dipropylene glycol monomethyl ether; ethyl acrylate; 2-nitro-p-phenylenediamine; o-toluidine; vinyl chloride; chlorodifluoromethane; 4-amino-2-nitrophenol; xylene.
Verlag Chemie GmbH, Postfach 1260/1280, 6940 Weinheim, Federal Republic of Germany, 12th supplement, 1986. Full work: 4 vols. Bibl.

CIS 86-1573 Maximum allowable workplace concentrations and biological limit values, 1986
Maximale Arbeitsplatzkonzentrationen und biologische Arbeitsstofftoleranzwerte 1986 [in German]
Definition of the notion of maximum allowable workplace concentration (MAK) and discussion of its basis and usefulness (limited, in the case of pregnant workers), of verification of compliance, of the relation of TWA-based MAK to peak exposures, and of the special problems posed by mixtures, allergens, and substances absorbed by the skin. List of 350 substances with their chemical formulae and MAKs in ppm and mg/m³. Compared to the 1985 list, there are substances added and some changes have been made. More detailed information is given on known or suspected carcinogens (about 140, including drugs and passive smoking), dusts (especially quartz and asbestos), organic peroxides, petrol (gasoline), turpentine, pyrolysis products and cutting fluids. "Indicative technical concentrations" (TRK) are given for about 30 carcinogens or mutagens the use of which in industry is unavoidable and for which no MAKs have been determined. "Biological limit values" (BAT) are defined and listed for 23 substances, including nitrobenzene, pentachlorophenol and toluene.
Senatskommission zur Prüfung gesundheitsschädlicher Arbeitsstoffe, Deutsche Forschungsgemeinschaft, Kennedyallee 40, 5300 Bonn 2, Federal Republic of Germany, 10 July 1986. Mitteilung XXII. 104p.

1985

CIS 97-1774 Regulation concerning passive smoking in the workplace 1985 [Iceland]
Reglur um tóbaksvarnir á vinnustöðum [in Icelandic]
This Regulation specifies measures for the prevention of harmful effects due to passive smoking in the workplace.
Administration of Occupational Safety and Health, P.O. Box 10120, 130 Reykjavik, Iceland, 1988. 1p.

CIS 88-874 International Agency for Research on Cancer - Annual Report 1985
Centre international de recherche sur le cancer - Rapport annuel 1985 [in French]
Occupational hazards considered in the report were: inhalable particles; registry of people exposed to dioxin-contaminated substances; epidemiology on the effects of pesticides in Colombia; passive smoking; case-control studies network; studies in Trieste and Singapore; N-nitroso compounds; opium, betel nut and other alkaloids; analgesic-related renal diseases; ochratoxin A; the IARC monographs; an occupational cancer review.
International Agency for Research on Cancer, 150 cours Albert-Thomas, 69372 Lyon Cedex 08, France, 1985. 240p. Illus. Bibl.

CIS 88-277 Henschler D.
Passive smoking in the workplace
Passivrauchen am Arbeitsplatz [in German]
Chapter contained in the loose-leaf collection "Toxicological and Medical Basis for the Establishment of Maximum Permissible Concentrations". (Gesundheitsschädliche Arbeitsstoffe - Toxikologische-arbeitsmedizinische Begründung von MAK-Werten). The levels of the main airborne substances which might been inhaled by passive smokers are given in a table. Results of epidemiological studies and possible mutagenic and carcinogenic effects of passive exposure to cigarette smoke are discussed.
Verlag Chemie GmbH, Postfach 1260/1280, 6940 Weinheim, Federal Republic of Germany, 11th. supplement. 1985. Vol.1. 26p. 95 ref.

CIS 88-260 Pollution of workplaces by tobacco smoke and deterioration of working conditions
Pollution des lieux de travail par la fumée du tabac et atteintes aux conditions de travail [in French]
Summary of the proceedings of and the proposals made at the 12th Congress of LCFTP (Versailles, France, 20-21 Apr. 1985). Two points were emphasised: the right of every worker to breathe air that is as little polluted as possible, and the scientific evaluation of the health hazards associated with passive smoking.
Ligue Contre la fumée du tabac en public, LCFTP, 14 rue du Petit Ballon, 68000 Colmar, France, 1985. 5p.

CIS 86-707 Sorso M., Einistö P., Husgafvel-., Jörventaus H., Kivistö H., Peltonen Y., Tuomi T., Valkonen S., Pelkonen O.
Passive and active exposure to cigarette smoke in a smoking experiment
6 volunteer female habitual smokers were exposed during a 2-week experimental period to cigarette smoke, both actively and passively in an exposure chamber where the ambient concentrations of carbon monoxide, particles, and aldehydes were monitored. Samples of blood and urine were taken from each subject after 3 nonsmoking days and after each day of active or passive smoking. The only parameters showing an increasing trend after passive exposure, as compared with nonsmoking samples, were urinary mutagenicity and plasma cotinine, the main metabolite of nicotine.
Journal of Toxicology and Environmental Health, 1985, Vol.16, No.3-4, p.523-534. Bibl.

CIS 86-134 Maximum allowable workplace concentrations and biological limit values 1985
Maximale Arbeitsplatzkonzentrationen und Biologische Arbeitsstofftoleranzwerte 1985 [in German]
The 1985 edition of this handbook (for previous edition, see CIS 84-1640) includes information on 350 substances (ca. 130 recognised or suspected carcinogens, including drugs and passive smoking). "Biological limit values" (BAT) are provided for 21 substances, with new or changed information on 8 substances or classes of substances: acetylcholinesterase inhibitors, aluminium, aniline, halothane, hydrogen fluoride and inorganic fluorides, parathion, tetrachloromethane, trichloroethylene.
Senatskommission zur Prüfung gesundheitsschädlicher Arbeitsstoffe, Deutsche Forschungsgemeinschaft, Kennedyallee 40, 5300 Bonn 2, Federal Republic of Germany, 16 July 1985. Mitteilung XXI. 100p.

CIS 86-133 Henschler D.
Passive smoking in the workplace
Passivrauchen am Arbeitsplatz [in German]
Scientific background of the decision to include a new paragraph on "passive smoking" in the chapter on carcinogenic substances of the 1985 MAC list of the Federal Republic of Germany. The Commission for the Study of Harmful Industrial Substances analysed the conditions of exposure to tobacco smoke by the non-smoker, it reviewed the results of epidemiological research and it evaluated the results of research in man and in animals involving mutagenic and carcinogenic substances in tobacco smoke. In conclusion, it is stated that tobacco smoke contains a mixture of substances harmful to the non-smoker in the workplace, that tobacco smoke is probably the harmful substance to which workers are most widely exposed and that tobacco smoke inhaled passively should be considered on the same basis as other harmful and carcinogenic substances or mixtures.
Senatskommission zur Prüfung gesundheitsschädlicher Arbeitsstoffe, Deutsche Forschungsgemeinschaft, Kennedyallee 40, 5300 Bonn 2, Federal Republic of Germany, 1985. 34p. 95 ref.

1984

CIS 88-269 The chronic consequences of smoking: Chronic obstructive lung disease - A report of the Surgeon General
Chapter 7 of this major report made to the US Congress is on passive smoking, including information on the measurement of exposure, acute physiological response of the airway to smoke in the environment, symptomatic response to chronic passive cigarette smoke exposure in healthy subjects, pulmonary function in adults exposed to involuntary cigarette smoke and the effects of passive smoke exposure on people with allergies, asthma and chronic obstructive lung disease.
Department of Health and Human Services, Public Health Service, Office on Smoking and Health, Rockville, MD 20857, USA, 1984. 545p. Illus. Bibl. Index.

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